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Experiments with a hypoxia tolerant bivalve

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Post-mortem findings showed higher GSH levels in the caudate, cerebellar, and frontal regions (Tong et al., 2016)

These systems may prevent quinones/hydroquinones from redox cycling reactions, the formation of lipid peroxidation products, GSH depletion, and the inactivation of signaling molecules such as hydroxynonenal and other molecules derived from the lipid peroxidation process capable of triggering apoptosis and ferroptosis (Dodson et al