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N.FuJ.NallD.RodovaM.ShankarS.SrivastavaR

Essentially, p62 interacts with the Nrf2-binding site on Keap1, a component of Cullin-3-type ubiquitin ligase for Nrf2, and when SQSTM1/p62 accumulates due to autophagy deficiency, it will compete with the interaction between Nrf2 and Keap1, resulting in stabilization of Nrf2 and transcriptional activation of Nrf2 target genes

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Adenosine is known to play a role in pain transmission, and its deficiency may exacerbate pain symptoms in FM patients (Fais et al., 2013)