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The administration of Fer-1 markedly prevented ultrastructural and morphological alterations in Raji cells, such as increased density of mitochondrial membranes and incomplete mitochondrial cristae, in Raji cells (Fig

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As a follow-up to Lehninger's work several years later, Nishikimi and co-workers observed that the gene that codes for gulonolactone oxidase is actually present in humans, but is not active due to the accumulation of several mutations that turned it into a non-functional pseudogene (Nishikimi & Yagi 1991)