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It is noteworthy that even under the cell-free inside-out configuration, membrane stretch was found to directly activate single-channel activity of TRPM7 by increasing the open probability ( P o ) (Numata et al., 2007a)

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Therefore, our studies add novel insights into a growing body of evidence implying that a combination of virus and host factors such as residual immune activation, hematopoietic dysregulation, and autoimmune phenomena could contribute to LC syndrome