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From a pathophysiological perspective, dietary fructose promotes DNL, impairs fatty acid oxidation, and triggers hepatic inflammation, thereby clearly fueling hepatic insulin resistance (reviewed in [99])

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National Institutes of Health, Bethesda, MD, USA) was used to identify and quantify the number, width, and length of the tubes

However, the heterogeneity in treatment protocols, such as formulation and dosage of omega-3s used, suggests the need for standardized recommendations in clinical practice [168,169,170,171,172,173,174,175,176,177,178,179]