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Gohda, T

Possibilities for therapies targeting cholesterol uptake in breast and ovarian cancers Cancers often upregulate the key cholesterol uptake molecules such as NPC1, NPC1-like 1 protein (NPC1L1), LDLR, SR-B1, and proprotein convertase subtilisin/kexin like 9 (PCSK9) making them the most logical targets of possible pharmaceutical interventions

These processes are catalyzed by specific enzymes such as the cytochrome P-450 enzyme system (CYP-450) and transferases (Figure 2)

While mRNA levels may not directly reflect protein presence, this finding argues against a potential simple explanation for DTT resistance in our mutants, whereby rips-1 mutation results in the loss of DTT uptake