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We observed that (i) MG132 treatment upregulated BAG3, consistent with a previous report 62 , and promoted the association between endogenous BAG3 and FLAG-CTIF (Supplementary Fig

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The presence of DTT-cysteine adducts at C41 and C108 are perhaps counter-intuitive, as C110 is implicated as the major site of protein cross-linking 25

The mechanism is twofold: GHRH activates its cAMP pathway while GHRP partially blocks somatostatin and activates the calcium pathway