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Antioxidants in the fight against atherosclerosis: is this a dead end

Collectively, these results indicate that hippocampal astrocytic Hb, rather than CA1 pyramidal neuronal Hb, is necessary for the antioxidative action of KDS12025, effectively alleviating astrogliosis and hippocampus-dependent memory impairment in an animal model of AD

390 Other clinical trials combining p53-MVA and the anti-PD1 antibody pembrolizumab are ongoing (NCT02432963 and NCT03113487)

During metabolic stresses such as increased oxidative phosphorylation or changes in cytoplasmic homeostasis that induce ER stress, the UPS will be compromised, which would most likely result in a reduction in BACE1 degradation by the UPS and lysosomes