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One cause of such a cytokine storm is an imbalance of the ratio between ACE1 (the pro-inflammatory mediator) and ACE2 (the anti-inflammatory mediator)

Two main pathways can trigger this process, the intrinsic, also called mitochondrial pathway, when apoptosis is initiated by the internal signals of mitochondrial origin, and the extrinsic when apoptosis is activated by external stimuli [16]

FUS , similar to SOD1 , is not a TDP-43 proteinopathy [8]

People who are heterozygous for this variant have reduced caffeine metabolism compared with those with alternative genotypes [40] and it may have a similar effect on TMA metabolism