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Taken together, GH excess associated with poorly controlled T1DM induces podocyte injury, characterized by podocyte hypertrophy, apoptosis, dedifferentiation of podocytes (epithelialmesenchymal transition) and/or cross-linking of the basement membrane resulting in increased podocyte permeability to albumin and detachment of podocytes from the glomerular basement membrane (Fig

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It supports lean muscle development, fat loss, improved sleep, and recovery with minimal side effects

Elevated GH/IGF-1 promotes lipolysis , preferentially targeting visceral adipose tissue (VAT), while preserving or modestly increasing lean body mass 3