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The present-day research also focuses on using antioxidants for the chemoprevention of different types of cancer, including brain cancer, as antioxidants may reduce tumor growth and interfere with carcinogenesis [4]

In support of our reasoning, (1) deletion of the regulatory domain prevents filament formation (Supplementary Fig

Evidence also implicates oxidative stress as a potential regulator of NF- B transactivation by MAPKs (in particular extracellular signal-regulated kinase 1/2 (ERK1/2) and p38) [30] which could lead to increased proteolysis in muscle [31]