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Type IV pili, conjugation genes, and plasmid-borne type III effectors were induced early in both niches, suggesting their niche-independent role in initial establishment

Review of pharmacological therapies in fibromyalgia syndrome

Human Cu/Zn superoxide dismutase (SOD1) overexpression in mice causes mitochondrial vacuolization, axonal degeneration, and premature motoneuron death and accelerates motoneuron disease in mice expressing a familial amyotrophic lateral sclerosis mutant SOD1

Collectively, these results demonstrated that Crizotinib caused liver ferroptosis by inhibiting Nrf2, a potential therapeutic target for its hepatotoxicity