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Intriguingly, the G3BP1 HKO MASLD model male mice exhibit dysregulated autophagy, and biochemical analyses demonstrated that G3BP1 promotes autophagosome-lysosome fusion through direct interactions with the SNARE proteins STX17 and VAMP8

Untreated TBI mice displayed statistically significant, approximately 4-fold and 2.5-fold increases in the number of fields with single and multiple Fluoro-Jade C-positive foci, respectively, in comparison to the Sham control group (Figures 11C and 11D)

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The recombinant FhGST-S1 enzyme was shown to be active, following the conjugation of substrates 1-chloro-2,4-dinitrobenzene (CDNB) and reduced glutathione according to an adaptation of the method described previously 25