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i.p.) prior to injection of the metabolic inhibitor 3-nitropropionic acid (3-NPA) in adult Sprague Dawley rats attenuated the 3-NPA induced increase in expression of the uncoupling protein UCP-2 and dopamine D(1) receptor genes in striatum that were upregulated in response to impaired metabolism [87]

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It was suggested that PGE 1 confers protection against endometriosis by attenuating the secretion of TNF-, IFN- and interleukins (IL-2, IL-4 and IL-6), while PGE 2 induces cytokine release and thus endometriosis through its apoptotic and inflammatory functions [19, 46]

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