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c-MET can either act as an immunosuppressive agent by negatively affecting dendritic cells (DC) and T lymphocytes or can stimulate immune response by promoting recruitment of DC, B-cells and T-lymphocytes [70]

Consequently, modulation of the AMPK/SIRT-1/m-TOR pathway has been proposed as an attractive approach for promoting autophagy and enhancing neuronal survival (Curry et al