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doi: 10.1097/01.jcp.0000095340.32154.c6

LongYQiuJZhangBHePShiXHeQet alPharmacological vitamin C treatment impedes the growth of endogenous glutamine-dependent cancers by targeting glutamine synthetase

Furthermore, even in p53-mutant conditions, the cell cycle can be blocked as p27 levels increase through p53-independent mechanisms, including reduced AKT-mediated degradation and FoxO transcription factor activation