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The role of DAB2IP in suppressing the inflammatory response is directly evidenced by upregulating the expression of pro-inflammatory chemokines, including C-X-C motif chemokine ligand 9 (CXCL9), CXCL10, and CXCL11, which exacerbate neointimal plaque formation

However, frequent blood transfusions can also lead to iron overload, damaging organs such as the liver and heart

Furthermore, the gut flora alters the bile acid pool, influencing glucose and energy homeostasis

Activation of dietary or supplemental vitamin B2 (riboflavin) requires a series of activation steps involving Thyroid Stimulating Hormone (TSH), thyroid hormone (T4), deiodinated T4 - triiodothyronine (T3), activation of riboflavin to FMN and finally modification of FMN to form FAD