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A high-cholesterol diet exacerbates mitochondrial dysfunction, ER stress, and chronic inflammation, all of which contribute to hepatic injury and the transition from steatosis to fibrosis and HCC [26, 30]

In research published in the journal Nature , Chen examined lung damage in animal models and identified age as a significant factor associated with an increased likelihood of fibrosis

There was a significant interaction with smoking status in the uni-variable model as well as model b ( p = 0.074 and p = 0.079, respectively), but not with sex ( p = 0.462 and p = 0.606, respectively) (Table 2)