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More importantly, this study further showed that SLC7A11 as a candidate gene of ferroptosis in HH, and suggested that the ROS-Nrf2-ARE axis was responsible for the upregulated SLC7A11, which may be a possible compensatory mechanism to protect against iron overload-induced ferroptosis in HH [25]

Furthermore, Western blot results also indicated that the supernatant of tumor cells treated with NP4 promoted TRPM2 protein expression in macrophages (Fig

Thus, it is suggested that the opposite regulation of these branches enhances production of isoflavones that act as antioxidants and antimicrobial compounds vs