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Furthermore, knockdown of GhODO1 impaired JA-mediated defense signaling and JA accumulation (Zhu et al., 2022)

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Simultaneously, the more toxic phytolaccoside H can undergo hydrolysis of the C 28 -ester glycosidic bond and release one glucose molecule, yielding the relatively less toxic phytolaccoside A, thus achieving the conversion of highly toxic constituents into less toxic ones

In FRTL-5 cells grown in the absence of TSH, NIS activity declines to a minimum level and can be restored by the addition of TSH to the medium or by treating the cells with dibutryl-cAMP or other agents that increase the intracellular concentration of cAMP (Pekary et al