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Virus-hijacked host cellular factors Clinical outcomes of COVID-19 are directly related to the ability of the SARS-CoV-2 pathogen to hijack host metabolic machinery as well as cellular factors of an infected individual, for viral invasion and internalization, followed by intra-cellular replication to assemble and release multiple viral copies for ultimate propagation/transmission

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The heterogeneity in this study was substantial, and we speculate that most of the heterogeneity may have come from clinical sources

These effects were also associated with significant reductions in measures of oxidative stress, including decreased lipid peroxidation and increased total reduced glutathione level