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Studies involving approximately 900 participants consistently demonstrated fat loss without changes in IGF-1, preserved glucose homeostasis despite fat reduction, absence of anabolic tissue growth effects, and safety profile comparable to placebo

56 These results highlight the possibility that genetic variation may have significant effects on susceptibility toward cancer

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These data support the notion of the inability of UPR to induce stress responses, favoring the buildup of prooxidant species and the damage of biological components