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The viral infection could also cause rapid T lymphocytopenia with functional impairment of T cells, which may onset OxS, pro-inflammatory state, cytokine production, and apoptosis 166,167

We previously demonstrated that melanocytes and fibroblasts from vitiligo patients displayed intrinsic alterations related to metabolic impairment that, when persistent, may lead to a senescent phenotype 1,22

Results Our findings revealed that oxidative stress primarily affected the endoplasmic reticulum (ER), causing notable alterations in the expression of ER-associated proteins, redox-responsive enzymes, chaperones, and sialyltransferases