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Regulation of lpha-synuclein gene (SNCA) by epigenetic modifier TET1 in Parkinson disease

DOI: 10.2119/molmed.2012.00332 Dahan A et al

It has been shown that the accumulation of advanced glycation-end products (AGEs) in alveolar macrophages contributes to IPF, and that inhibition of the formation of AGEs prevents the disease in animal models [42, 43]

In rats bearing this tumor, induction of type-1-like diabetes with alloxan was followed in up to 90% of cases by rapid onset of remission, whereas treatment with insulin and especially combined treatment with insulin and glucose stimulated tumor growth considerably