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Collectively, these findings support a bidirectional relationship in which sleep promotes mitochondrial redox homeostasis, while mitochondrial oxidative status shapes sleep quantity, quality, and circadian stability, processes with broad implications for aging and neurodegenerative diseases

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The increased number of hepatocytes resulting from the hyperplastic response is generally believed to remain at the elevated level, having established a new set point of adaptive function, until the xenobiotics stimulus is removed