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All this suggests clock genes, especially BMAL1, as a strong candidate in the pathogenesis of the possible COVID-19-induced acceleration of aging, with a potential role for iron disruption and ferroptosis

PMID: 7028698

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this, in turn, leads to accentuated elevations in blood concentrations of tHcy and MMA, and exacerbation of neurological sequelae due to vitamin B-12 deficiency in the brain and peripheral nervous system To test these and other predictions, in addition to human studies, it will be important to develop better animal models with closer similarity to the physiologic and clinical features of vitamin B-12 deficiency than rodent models currently provide