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Importantly, these early-phase clinical investigations have not been restricted to HCC

Cells 10 (1), 66

doi: 10.1016/j.cmet.2018.04.010 139 TavakoliA.AkhgarjandC.AnsarH.HoujaghaniH.KhormaniA.DjafarianK.et al

Since the capacity of GR1 in the Arabidopsis cytosol is large and far from limiting under normal growth conditions 63 , inhibition of GR1 by (hyper)oxidation of catalytic or regulatory cysteine residues provide a plausible hypothesis 64 either as a result of oxidative damage or as a mechanism to safeguard NADP redox status, similar to what has previously been observed for peroxiredoxins 50,65