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The net effect: adipocytes become metabolically sluggish, oxidizing less fat, storing more energy, and resisting signals that would normally promote energy expenditure

Mitochondrial dysfunction, a key feature of AD, further exacerbates ROS production, impairs ATP generation, disrupts calcium homeostasis, and ultimately induces neuronal apoptosis (Butterfield and Halliwell, 2019)

While some patients tolerate treatment well when appropriately evaluated, safety depends on individual health status, medications, and clinical judgment

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