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No differences were found between 2.5 mg/kg MOR-treated groups when compared to SAL-treated controls for each of ALP pretreatments (0.5, 1.0 mg/kg)

In particular, lack of early response appears to be strongly indicative of subsequent nonresponse [2], failure to achieve remission [3], and higher rates of treatment discontinuation [4]

ALCAR and KET also activated brainstem nuclei (locus coeruleus, diagonal band, and raphe nuclei) which send aminergic projections to cortical areas and spinal dorsal horns that are involved in pain and mood control (Khan and Stroman, 2015)

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