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Formation of specific liver fibrosis patterns is a dynamic and multi-cellular process, which is difficult to uniquely explain by experiments both, in vivo and in vitro, and therefore not sufficiently understood

At normal paracetamol dose the liver is able to inactivate this potentially damaging NAPQI by reaction with a substance called glutathione, which is synthesised in the liver

Because oligodendrocytes do not have the ability to regenerate following injury, a large number of these cells die rapidly (within 3 hours) of ischemic stroke as a result of toxic levels of ATP and glutamate release [32]