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As mentioned above, CARS, like other aminoacyl-tRNA synthetases, is primarily involved in cysteinyl-tRNA production utilizing CysSH

This response may be part of an emergency response with the sudden onslaught of oxidants from the CS exposure or it may be the depletion of GSH during the CS exposure combined with the increase in oxidants from CS that acts as a trigger to initiate this response

In rat hindlimb ischemia models, this mechanism accelerated blood flow recovery and increased vessel density in oxygen-deprived tissues

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