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Neuroimmune communication via calcitonin gene-related peptide (CGRP) and its coreceptor RAMP1 promotes macrophage recruitment and phenotypic shifts toward a pro-endometriosis state, characterized by impaired efferocytosis and enhanced support of endometrial cell growth ( Natural killer (NK) cell function is severely compromised, with reduced cytotoxicity of the CD56dimCD16+ subset in peripheral blood and peritoneal fluid, enabling immune escape of ectopic cells ( T-cell subsets are dysregulated, with increased Th2, Th17, and regulatory T (Treg) cells in the peritoneal microenvironment ( 2.2.2 Immune checkpoint molecules Immune checkpoint pathways are perturbed, contributing to immunosuppression

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627 Traditionally, MDD was attributed to neurotransmitter alterations or brain-derived neurotrophic factor disorders

To validate the occurrence of ferroptosis in the mouse model, proteins from renal proximal tubular tissues were analyzed using Western blot