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While various studies have proven that activation of the Nrf2-HO-1 pathway can prevent ISO-induced myocardial injury 36,37 , the specific mechanisms and potential of natural substances in modulating this pathway remain an ongoing area of research

Its influence extends to ameliorating microcirculatory disturbances seen in bone necrosis [1] and positively adjusting serum physiological markers, including total antioxidant capacity and lactate dehydrogenase [14], alongside inflammatory markers like the erythrocyte sedimentation rate [13]