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The significant connection between vitamin B5 and energy metabolism implies its potential to influence the pathophysiological processes of LLD by enhancing mitochondrial health and boosting energy production

doi: 10.1152/ajpgi.00352.2011

Similarly, in neuron-related studies, pharmacologically activating NCLX (using the phosphodiesterase 2 inhibitor Bay 60-7550) to enhance calcium efflux can protect neurons from excitotoxic injury and improve learning and memory in mice [54]