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Collectively, these agents converge on the Nrf2, SIRT1, and AMPK signaling networks to restore redox homeostasis, prevent mitochondrial dysfunction, and inhibit oxidative stressinduced apoptosis, ultimately improving follicular viability and oocyte developmental competence during cryopreservation and transplantation

J Control Release 348:321334

The honest summary: the mechanistic case for BPC-157 supporting tendon and ligament repair is well-documented in animal and cell-culture research, and the rationale for pairing it with TRT is backed by real human data showing testosterone therapy raises tendon injury risk