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This effect is mechanistically distinct from galangin, which suppresses aberrant NLRP3 activation in ovarian cancer xenograft mouse models, downregulating both NLRP3 expression and IL-1 maturation ( 3.4.2 Inhibition of the NF-B pathway Nuclear factor-B (NF-B) is a transcription factor highly associated with inflammation, mainly composed of the p65 subunit and the p50 subunit

Starvation, together with the SOS response, mediates high biofilm-specific tolerance to the fluoroquinolone ofloxacin

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This issue is severely exacerbated when coexisting biomarkers or exogenous drugs have closely overlapping redox potentials [166]