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AhR activation can inhibit inflammasome (such as NLRP3) activation, reduce the production of pro-inflammatory factors like IL-1, and promote IL-22 secretion, which has a protective effect on hepatocytes and can reduce oxidative stress and steatosis

Based on the available data we propose IGF-1 as a key hormone in the pathophysiology of metabolic syndrome

These uncharged weak bases reach parietal cells from the blood and diffuse into the secretory canaliculi, where the drugs become protonated and thereby trapped