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MOTS-C is a mitochondrial-derived peptide with academic interest in metabolic regulation and almost no clinical trial data

T h 17 and T h 22 cells, meanwhile, have been observed to display atheroprotective and proatherogenic effects and therefore warrant continued investigation

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Taken together, these mechanistic and clinical threads support a model in PCOS whereby platelet activation and PMP release prime neutrophils for NETosis, and NETs provide a prothrombotic scaffold that recruits and activates additional platelets and coagulation factors, producing an escalating thrombo-inflammatory loop with potential consequences for ovarian microvasculature, endometrial receptivity, and long-term cardiovascular risk [77]