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The mechanisms underlying these effects in alveolar epithelial cells may involve ferroptosis inhibition via the Sirt1/Nrf2/Gpx4 signaling pathway (Fig

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OCA treatment ameliorated high-fat diet (HFD)-induced dysplasia, potentially by reducing deleterious secondary BAs (e.g., DCA, TDCA), decreasing microbial bile salt hydrolase (BSH) expression, and favorably reshaping the tumors immune landscape (128), thereby potentially reversing an immunosuppressive TIME

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