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However, the cellular stress of the upstream events, i.e., reactive metabolite formation, GSH depletion, and protein adducts formation, is still present and this stress may trigger apoptosis in a limited number of cells (Figure 1)

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NAC also maintains mitochondrial redox homeostasis by activating the SIRT3-SOD2-GPX4 signaling pathway, thus reducing the ferroptosis rate in the context of DN