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In any case, this context leads to a vicious cycle which compromises ATP production and neuronal viability (Figure 2)

Suppression of SNAIL2, a zinc finger protein member of the SNAIL superfamily of epithelial-mesenchymal transition-inducing transcription factors (EMT-TFs) commonly overexpressed in pancreatic cancer patients, restored sensitivity of pancreatic cancer cell lines KLM1 and KMP5 to chemotherapy drug gemcitabine through an IGF binding protein 2 (IGFBP2)-dependent mechanism in vitro and in vivo (50)

Microbial tryptophan metabolites regulate gut barrier function via the aryl hydrocarbon receptor