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The amino acid sequence of rat liver G6PD shows 94% homology to the human sequence and provides evidence that the N-terminal amino acid is N -acetylalanine, which must result from posttranslational cleavage of the N-terminal methionine

Knockdown of each gene diminished GSH levels, and knockdown of Idh2 and Me1 also increased GSSG levels, thus elevating the GSSG/GSH ratio (Figure 6D)

iii) The lumen is segmented by applying distance transform on the centerline

in contrast, only Cu chelator treatment show potent rescue effect on such excess Cu 2+ -induced cell death, suggesting that cuproptosis is distinct from other forms of cell death