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These approaches are particularly attractive because of their potential to modulate several cardiovascular risk factors simultaneously through different mechanisms of action [3, 5, 6]

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In the case of adhI , its promoter was shown to be activated by both formaldehyde and methanol, although induction by formaldehyde occurs more rapidly and at a lower concentration ( adhI is controlled by two separate, two-component regulatory systems: (i) AfdRS, which activates transcription in the presence of formaldehyde, and (ii) RfdRS, which is thought to act as a repressor, although the signal for derepression is unknown ( afdR and afdS are organized within an operon, as are rfdR and rfdS

[note: small fluctuations in ALT (e.g