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In summary, dysbiosis might contribute to neurodegeneration by disrupting metabolic homeostasis reducing beneficial metabolites (SCFAs, vitamins) and increasing potentially neurotoxic ones (e.g

doi: 10.3389/fmed.2018.00061

polygyrus -derived Hpb glutamate dehydrogenase (GDH), which reduced allergic airway inflammation in mice by inducing a switch from pro-inflammatory to anti-inflammatory eicosanoids (e.g., prostaglandins)

158 Available findings have allowed us to speculate that the biological activities of oxidized AAT may depend on the magnitude of AAT oxidation, in a manner similar to that discussed for HSA (Figure 3)