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Effect of developmental NMDAR antagonism with CGP 39551 on aspartame-induced hypothalamic and adrenal gene expression

Lack of such regulatory mechanisms, as described with our gsh2 mutant, resulted in enhanced phagocytosis, lack of melanin, and impaired neurotropism

7.3 Applicable populations and disease stages for exercise intervention mechanisms The exercise-induced mechanisms outlined above are chiefly derived from studies conducted on patients with impaired pulmonary function, including those in the acute phase (e.g., early rehabilitation following acute respiratory distress syndrome), recovery phase (post-discharge pulmonary rehabilitation), and chronic phase with persistent functional limitations (e.g., chronic obstructive pulmonary disease, pulmonary fibrosis)

Benveniste H, Liu X, Koundal S, Sanggaard S, Lee H, Wardlaw J