Vol. XVIII · Free shipping $75+ · Read the collection
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Description

Furthermore, inhibition of SLC7A11 via genetic or pharmacological targeting attenuates mutant-KRAS associated tumor growth in xenograft models via induction of lipid peroxidation and ferroptosis ( Slc7a11 deficient mice that oncogenic KRAS-driven pancreatic adenocarcinoma (PDAC) tumor growth is significantly impaired (78)

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