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Together, these findings suggest that KYN-driven oxidative stress is not merely an epiphenomenon of depression but a mechanistic contributor to mitochondrial dysfunction, neuroprogression, and systemic comorbidity, highlighting redox modulation as a critical interface between inflammation, KP activity, and depressive pathology

The tissue microarrays (TMAs) were constructed as described previously ( H -score, a combination of the proportion of positive cells and the staining intensity, was applied to assess the results of IHC

220 Therefore, it is highly important to explore the relationships among lipid and glucose metabolism, mitochondria metabolic function and ferroptosis in metabolic diseases such as diabetes, hyperlipidemia, and obesity

doi: 10.1186/s12974-018-1324-0 90 MohammadiM