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Table 5 exhibits a summarized form of the implications of nanomedicine on PD

However, in resistant patients, oxidative stress causes a conformational change in Keap1, leading to the release of NRF2, which translocates to the nucleus and binds to antioxidant response element (ARE), activating the expression of downstream antioxidant genes, including GPX4, NQO1, HO-1 and SOD1 [33], and promoting the synthesis of GSH, as illustrated in Fig

Surprisingly, malignant cells can evade the deleterious effect of excessive iron and require high amounts of these reactive ions for their proliferation (Pfeifhofer-Obermair et al., 2018)