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(4) In chemotherapy-induced premature aging (a mouse model of cachexia), FOXO4-DRI partially reversed the metabolic aging phenotype

It's not just labeling semantics or marketing language

In line with the IR-senescence data, low effective doses of FOXO4-DRI were well tolerated in normal IMR90 compared to ABT-737, while being very potent against Doxorubicin-senescent cells at higher doses (Fig

Some experimental models indicate that GHK-Cu may regulate fibrosis-related gene pathways, suggesting a possible role in conditions involving abnormal tissue scarring