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Introducing the disulfide between Fc (A330C) and FcRIIIa (I106C) yields stable, covalently linked complexes that do not require post-expression modification, are compatible with standard mammalian expression, and support payloads such as anti-CD3, anti-CD28, IL-2, and protease-activated IL-2

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After oral administration, NAC is primarily metabolized in the liver to cysteine, which enters the bloodstream [110]